CACNA1H monoclonal antibody, clone S55-10 View larger

CACNA1H monoclonal antibody, clone S55-10

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Data sheet of CACNA1H monoclonal antibody, clone S55-10

BrandAbnova
Product typePrimary antibodies
ReactivityHuman,Mouse,Rat
Host speciesMouse
ApplicationsWB,IP,ICC,IF

More info about CACNA1H monoclonal antibody, clone S55-10

Brand: Abnova
Reference: MAB6650
Product name: CACNA1H monoclonal antibody, clone S55-10
Product description: Mouse monoclonal antibody raised against partial recombinant CACNA1H.
Clone: S55-10
Isotype: IgG1
Gene id: 8912
Gene name: CACNA1H
Gene alias: CACNA1HB|Cav3.2|EIG6|FLJ90484
Gene description: calcium channel, voltage-dependent, T type, alpha 1H subunit
Immunogen: Recombinant fusion protein corresponding to amino acids 1019-1293 (II-III loop) of human CACNA1H.
Protein accession: O95180
Form: Liquid
Recommend dilutions: Western Blot (1-10 ug/mL)
Immunohistochemistry (0.1-1.0 ug/mL)
Immunocytochemistry (0.1-1.0 ug/mL)
Immunofluorescence (1.0-10 ug/mL)
The optimal working dilution should be determined by the end user.
Storage buffer: In PBS, pH 7.4 (50% glycerol, 0.09% sodium azide)
Storage instruction: Store at -20°C.
Aliquot to avoid repeated freezing and thawing.
Note: This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
Product type: Primary antibodies
Host species: Mouse
Antigen species / target species: Human
Specificity: Detects ~260KDa. No cross-reactivity against Cav1.3.
Reactivity: Human,Mouse,Rat
Application image: MAB6650-49-223-1.jpg
Application image note: Immunofluorescence staining in human hippocampus with CACNA1H monoclonal antibody, clone S55-10 (Cat # MAB6650).
Applications: WB,IP,ICC,IF
Shipping condition: Dry Ice
Publications: Ca(V)1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism.Splawski I, Timothy KW, Sharpe LM, Decher N, Kumar P, Bloise R, Napolitano C, Schwartz PJ, Joseph RM, Condouris K, Tager-Flusberg H, Priori SG, Sanguinetti MC, Keating MT.
Cell. 2004 Oct 1;119(1):19-31.

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